PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17557286-8	2007	Furthermore, we could show that antiangiogenic effects of thalidomide are related to tumor-cell derived factors including vascular endothelial growth factor, basic fibroblast growth factor, hepatocyte growth factor and Il-8 some known and with, granulocyte colony stimulating growth factor and granulocyte macrophage colony stimulating growth factor, some new target molecules of thalidomide.	Thalidomide	58-69	fibroblast growth factor 2	Mus musculus	158-188	
9301478-1	1997	Thalidomide, when administered orally, is an inhibitor of angiogenesis in the basic fibroblast growth factor (bFGF)-induced rabbit cornea micropocket assay.	Thalidomide	0-11	fibroblast growth factor 2	Mus musculus	110-114	
9301478-2	1997	We now show in the mouse that thalidomide given intraperitoneally but not orally significantly inhibits bFGF-induced and vascular endothelial growth factor (VEGF)-induced corneal neovascularization.	Thalidomide	30-41	fibroblast growth factor 2	Mus musculus	104-108	
9301478-4	1997	Experiments examining thalidomide"s enantiomers reveal-that the S(-)-enantiomer has the strongest antiangiogenic activity in VEGF-induced and bFGF-induced corneal neovascularization.	Thalidomide	22-33	fibroblast growth factor 2	Mus musculus	142-146