PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26481541-0	2015	Thalidomide can promote erythropoiesis by induction of STAT5 and repression of external pathway of apoptosis resulting in increased expression of GATA-1 transcription factor.	Thalidomide	0-11	GATA binding protein 1	Homo sapiens	146-152	
27047649-0	2016	Thalidomide is more efficient than sodium butyrate in enhancing GATA-1 and EKLF gene expression in erythroid progenitors derived from HSCs with beta-globin gene mutation.	Thalidomide	0-11	GATA binding protein 1	Homo sapiens	64-70	
27047649-7	2016	RESULTS: Thalidomide and sodium butyrate increased GATA-1 and EKLF gene expression, compared to the non-treated control (P<0.05).	Thalidomide	9-20	GATA binding protein 1	Homo sapiens	51-57	
27047649-8	2016	CONCLUSION: Thalidomide was more efficient than sodium butyrate in augmenting expression of GATA-1 and EKLF genes.	Thalidomide	12-23	GATA binding protein 1	Homo sapiens	92-98	
27047649-9	2016	It seems that GATA-1 and EKLF have crucial roles in the efficient induction of HbF by thalidomide.	Thalidomide	86-97	GATA binding protein 1	Homo sapiens	14-20