PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34339726-8	2021	We also showed that being treated with thalidomide and palladium (II) complex inhibited phosphorylation of the signaling regulators downstream of the VEGFR2.	Thalidomide	39-50	kinase insert domain receptor	Homo sapiens	150-156	
16840205-1	2006	Thalidomide is a drug with anti-angiogenic, anti-inflammatory, immunomodulatory and anti-cancer properties that were found to inhibit the production of TNF-alpha in vitro, stimulate reactive oxygen species production, and inhibit VEGFR in acute leukemias.	Thalidomide	0-11	kinase insert domain receptor	Homo sapiens	230-235	
15741222-3	2005	Thalidomide induces the defect of major blood vessels, which is demonstrated by their morphologic loss and confirmed by the depletion of vascular endothelial growth factor (VEGF) receptors such as neuropilin-1 and Flk-1.	Thalidomide	0-11	kinase insert domain receptor	Homo sapiens	214-219