PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
14693056-2	2003	The aim of this research was to observe the effect of thalidomide on tumor homograft growth in mouse H22 model and to investigate the mechanisms involved and its curative possibility to hepatoma.	Thalidomide	54-65	histocompatibility 22	Mus musculus	101-104	
14693056-15	2003	CONCLUSION: Thalidomide can significantly induce apoptosis and inhibit angiogenesis in mouse H22 model when it is used at the beginning of carcinogenesis, but it has no obvious anti-angiogenic efficacy on the grown tumor.	Thalidomide	12-23	histocompatibility 22	Mus musculus	93-96	
14693056-16	2003	Thalidomide cannot inhibit VEGF mRNA expression of grafted H22 tumor in mouse.	Thalidomide	0-11	histocompatibility 22	Mus musculus	59-62