PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27096074-10	2016	RESULTS: Treatment of NaHS (a H2S donor) protected against H/R-induced apoptosis, cell damage, the expression of cleaved caspase-3 and cleaved caspase-9, the release of cytochrome c (Cyt c).	sodium bisulfide	22-26	caspase 9	Homo sapiens	143-152	
29466396-10	2018	Furthermore, hypoxia significantly increased the levels of multiple apoptosis associated proteins (Fas, Cytochromes C, Caspase 9 and cleaved-Caspase-3) in cells, which were eliminated by NaHS.	sodium bisulfide	187-191	caspase 9	Homo sapiens	119-128	
30912763-4	2019	NaHS (a H2S donor) exerted significant protective effects against H/R-induced cell damage, apoptosis, production of cleaved caspase-3 and caspase-9, and release of cytochrome c.	sodium bisulfide	0-4	caspase 9	Homo sapiens	138-147	
23799362-9	2013	Additionally, NaHS increased Bcl-2 expression and decreased the expression of Bax, Caspase-3 and Caspase-9 in a dose-dependent way.	sodium bisulfide	14-18	caspase 9	Homo sapiens	97-106	
26229588-5	2015	Supplementation of NaHS protected against H/R-induced apoptosis, the expression of cleaved caspase-3 and cleaved caspase-9, the release of cytochrome c (Cyt c), and mPTP opening.	sodium bisulfide	19-23	caspase 9	Homo sapiens	113-122	
22555844-4	2012	NaHS also reversed the upregulation of cleaved poly(ADP-ribose) polymerase and caspase 9 in 6-OHDA-treated cells.	sodium bisulfide	0-4	caspase 9	Homo sapiens	79-88	
18832435-5	2009	NaHS also prevented rotenone-induced p38- and c-Jun NH(2)-terminal kinase (JNK)-mitogen-activated protein kinase (MAPK) phosphorylation and rotenone-mediated changes in Bcl-2/Bax levels, mitochondrial membrane potential (DeltaPsi(m)) dissipation, cytochrome c release, caspase-9/3 activation and poly(ADP-ribose) polymerase cleavage.	sodium bisulfide	0-4	caspase 9	Homo sapiens	269-278