PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30251691-8	2018	In cultured VSMCs, high glucose level also reduced H2S level, upregulated the autophagy-related index and activated the AMPK/mTOR pathway, which were reversed by concomitant application of sodium hydrosulfide (NaHS, an H2S donor) or ALA.	sodium bisulfide	189-208	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	120-124	
30251691-8	2018	In cultured VSMCs, high glucose level also reduced H2S level, upregulated the autophagy-related index and activated the AMPK/mTOR pathway, which were reversed by concomitant application of sodium hydrosulfide (NaHS, an H2S donor) or ALA.	sodium bisulfide	210-214	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	120-124	
30251691-9	2018	The protective effect of NaHS or ALA was attenuated by rapamycin (an autophagy activator), 5-amino-1-beta-d-ribofuranosyl-imidazole-4-carboxamide (an AMPK activator) or PPG.	sodium bisulfide	25-29	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	150-154	
30251691-10	2018	In contrast, Compound C (an AMPK inhibitor) enhanced the effect of ALA or NaHS.	sodium bisulfide	74-78	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	28-32