PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31840352-1	2020	OBJECTIVES: Mixed lineage leukaemia protein-1 (MLL1) mediates histone 3 lysine 4 (H3K4) trimethylation (me3) and plays vital roles during early embryonic development and hematopoiesis.	H-2K(K) antigen	82-86	lysine (K)-specific methyltransferase 2A	Mus musculus	12-45	
31840352-1	2020	OBJECTIVES: Mixed lineage leukaemia protein-1 (MLL1) mediates histone 3 lysine 4 (H3K4) trimethylation (me3) and plays vital roles during early embryonic development and hematopoiesis.	H-2K(K) antigen	82-86	lysine (K)-specific methyltransferase 2A	Mus musculus	47-51	
31840352-11	2020	Mechanically, MLL1 transcriptionally regulated Myf5 by mediating H3K4me3 on its promoter.	H-2K(K) antigen	65-72	lysine (K)-specific methyltransferase 2A	Mus musculus	14-18	
31840352-13	2020	CONCLUSION: MLL1 facilitates proliferation of myoblasts and Pax7-positive satellite cells by epigenetically regulating Myf5 via mediating H3K4me3 on its promoter.	H-2K(K) antigen	138-145	lysine (K)-specific methyltransferase 2A	Mus musculus	12-16	
31789407-9	2020	The differentially expressed genes with the same trend in H3K79me2, H3K27ac and H3K4me3 alteration were identified as potential MLL-AF9 direct target genes.	H-2K(K) antigen	58-66	lysine (K)-specific methyltransferase 2A	Mus musculus	128-131	
31789407-9	2020	The differentially expressed genes with the same trend in H3K79me2, H3K27ac and H3K4me3 alteration were identified as potential MLL-AF9 direct target genes.	H-2K(K) antigen	80-87	lysine (K)-specific methyltransferase 2A	Mus musculus	128-131	
31570196-3	2019	Recently, the accumulation of p16INK4a-positive cells in the kidney has been considered a molecular feature of renal senescence, with the p16INK4a gene reportedly regulated by mixed-lineage leukemia 1 (MLL1)/WD-40 repeat protein 5 (WDR5)-mediated histone 3 lysine 4 trimethylation (H3K4me3).	H-2K(K) antigen	282-289	lysine (K)-specific methyltransferase 2A	Mus musculus	176-200	
31570196-3	2019	Recently, the accumulation of p16INK4a-positive cells in the kidney has been considered a molecular feature of renal senescence, with the p16INK4a gene reportedly regulated by mixed-lineage leukemia 1 (MLL1)/WD-40 repeat protein 5 (WDR5)-mediated histone 3 lysine 4 trimethylation (H3K4me3).	H-2K(K) antigen	282-289	lysine (K)-specific methyltransferase 2A	Mus musculus	202-206	
31570196-5	2019	MM-102 or OICR-9429, both MLL1/WDR5 protein-protein interaction inhibitors, and small interfering RNA (siRNA) for MLL1 or WDR5 suppressed the expression of p16INK4a in mice with ischemia reperfusion injury, accompanied by downregulation of H3K4me3 expression.	H-2K(K) antigen	240-247	lysine (K)-specific methyltransferase 2A	Mus musculus	114-118