PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18957819-2	2008	In the present study, we investigated the effects of SPH with angiotensin-converting enzyme (ACE) inhibitory potential on blood pressure and renal injuries in rats with N(omega)-nitro-L-arginine methyl ester hydrochloride (L-NAME)-induced hypertension.	sph	53-56	angiotensin I converting enzyme	Rattus norvegicus	93-96	
18957819-6	2008	The levels of kidney ACE activity, malonaldehyde, tumor necrosis factor-a and plasminogen activator inhibitor-1, and the expression of CYP4A decreased in the 5% SPH group.	sph	161-164	angiotensin I converting enzyme	Rattus norvegicus	21-24	
18957819-8	2008	These findings suggest that SPH might ameliorate the elevation of blood pressure and show renoprotective effects in nitric oxide (NO)-deficient rats, and one possible mechanism might be mediation via its ACE inhibitory activity.	sph	28-31	angiotensin I converting enzyme	Rattus norvegicus	204-207	
27681454-11	2016	The malonyldialdehyde (MDA) levels in organs and serum, serum ACE activity, and serum ANG-II concentration in SPH-treated SHR rats were significantly lowered (P < 0.05 or P < 0.01) as compared to their controls.	sph	110-113	angiotensin I converting enzyme	Rattus norvegicus	62-65