PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32562757-0	2020	Rab11 is essential for lgl mediated JNK-Dpp signaling in dorsal closure and epithelial morphogenesis in Drosophila.	dipalmitoylphosphatidylserine	40-43	basket	Drosophila melanogaster	36-39	
32562757-4	2020	The JNK-Dpp signaling in the dorsolateral epidermis, plays an instrumental role in guiding their fate during this process.	dipalmitoylphosphatidylserine	8-11	basket	Drosophila melanogaster	4-7	
32562757-6	2020	Here we show a probable mechanism via which lgl, a conserved tumour suppressor gene, regulates the JNK-Dpp pathway during dorsal closure and epithelial morphogenesis.	dipalmitoylphosphatidylserine	103-106	basket	Drosophila melanogaster	99-102	
32562757-9	2020	Our experiment suggests Rab11 to be interacting with lgl as they seem to synergize in order to regulate the core JNK-Dpp signaling pathway during dorsal closure and also during adult thorax closure process.	dipalmitoylphosphatidylserine	117-120	basket	Drosophila melanogaster	113-116	
28315838-8	2017	Further, we discovered that the JNK signaling pathway operated between Rab5 and Dpp, because simultaneously inhibiting the JNK pathway and Rab5 in cyst cells prevented both dpp transcription and germline tumor growth.	dipalmitoylphosphatidylserine	173-176	basket	Drosophila melanogaster	32-35	
28315838-8	2017	Further, we discovered that the JNK signaling pathway operated between Rab5 and Dpp, because simultaneously inhibiting the JNK pathway and Rab5 in cyst cells prevented both dpp transcription and germline tumor growth.	dipalmitoylphosphatidylserine	173-176	basket	Drosophila melanogaster	123-126	
22114909-0	2011	Excessive Dpp signaling induces cardial apoptosis through dTAK1 and dJNK during late embryogenesis of Drosophila.	dipalmitoylphosphatidylserine	10-13	basket	Drosophila melanogaster	68-72	
22114909-9	2011	The hyperactivated DJNK was attributed to be the cause of Dpp/DTAK1-induced apoptosis because overexpression of a dominant negative DJNK, basket (bskDN), suppressed cell death induced by Dpp or DTAK1.	dipalmitoylphosphatidylserine	58-61	basket	Drosophila melanogaster	132-136	
22114909-9	2011	The hyperactivated DJNK was attributed to be the cause of Dpp/DTAK1-induced apoptosis because overexpression of a dominant negative DJNK, basket (bskDN), suppressed cell death induced by Dpp or DTAK1.	dipalmitoylphosphatidylserine	187-190	basket	Drosophila melanogaster	19-23	
22114909-9	2011	The hyperactivated DJNK was attributed to be the cause of Dpp/DTAK1-induced apoptosis because overexpression of a dominant negative DJNK, basket (bskDN), suppressed cell death induced by Dpp or DTAK1.	dipalmitoylphosphatidylserine	187-190	basket	Drosophila melanogaster	132-136	
22114909-11	2011	CONCLUSIONS: We find that ectopic Dpp led to DJNK-dependent cardial apoptosis through the non-canonical TGF-beta pathway during late embryogenesis of Drosophila.	dipalmitoylphosphatidylserine	34-37	basket	Drosophila melanogaster	45-49	
22114909-9	2011	The hyperactivated DJNK was attributed to be the cause of Dpp/DTAK1-induced apoptosis because overexpression of a dominant negative DJNK, basket (bskDN), suppressed cell death induced by Dpp or DTAK1.	dipalmitoylphosphatidylserine	58-61	basket	Drosophila melanogaster	19-23	
17333257-2	2007	Its expression is dependent on the Jun N-terminal kinase (JNK) cascade, and it functions downstream of Jun regulating dpp expression in the leading edge cells.	dipalmitoylphosphatidylserine	118-121	basket	Drosophila melanogaster	35-56	
19270172-6	2009	At the molecular level, reduced Dpp signaling results in Brk upregulation that triggers apoptosis through activation of the JNK pathway.	dipalmitoylphosphatidylserine	32-35	basket	Drosophila melanogaster	124-127	
19270172-7	2009	Here we show that the transcriptional co-regulator dNAB is a Dpp target in the developing wing that interacts with Brk to eliminate cells with reduced Dpp signaling through the JNK pathway.	dipalmitoylphosphatidylserine	61-64	basket	Drosophila melanogaster	177-180	
19270172-7	2009	Here we show that the transcriptional co-regulator dNAB is a Dpp target in the developing wing that interacts with Brk to eliminate cells with reduced Dpp signaling through the JNK pathway.	dipalmitoylphosphatidylserine	151-154	basket	Drosophila melanogaster	177-180	
17333257-2	2007	Its expression is dependent on the Jun N-terminal kinase (JNK) cascade, and it functions downstream of Jun regulating dpp expression in the leading edge cells.	dipalmitoylphosphatidylserine	118-121	basket	Drosophila melanogaster	58-61	
11865058-2	2002	By a genetic screen of mutations affecting dorsal closure in Drosophila, we have now identified a multidomain protein, connector of kinase to AP-1 (cka), that functions in the DJNK pathway and controls the localized expression of dpp in the leading-edge cells.	dipalmitoylphosphatidylserine	230-233	basket	Drosophila melanogaster	176-180	
9224720-6	1997	Our results provide a causal link between the DJNK and Dpp pathways during dorsal closure.	dipalmitoylphosphatidylserine	55-58	basket	Drosophila melanogaster	46-50