PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32649211-10	2020	This suggests that NCX1 could be a biomarker to identify TNBC patients that might benefit from the clinical administration of a cardiac glycoside for anticancer indications.	Glycosides	136-145	solute carrier family 8 member A1	Homo sapiens	19-23	
23153195-11	2012	CONCLUSION: This investigation shows that the cardiac glycoside OUA is cytotoxic also for the lymphoma derived cell line U937 and that can activate a survival pathway in which are involved NCX and p38 MAPK.	Glycosides	54-63	solute carrier family 8 member A1	Homo sapiens	189-192	
16399865-12	2006	We conclude that expression of the NCX1.3 splice variant protects against severe Ca2+ overload, whereas NCX1.1 promotes Ca2+ overload in response to cardiac glycosides and ischemic challenges.	Glycosides	157-167	solute carrier family 8 member A1	Homo sapiens	104-108	
8615321-1	1996	Cardiac glycosides and Na+ -channel activators increase intracellular Na+ and thereby enhance the transport rate of the sarcolemmal Na+/Ca2+ exchanger.	Glycosides	8-18	solute carrier family 8 member A1	Homo sapiens	132-150	
8615321-2	1996	We tested the hypothesis of whether increased expression of the Na+/Ca2+ exchanger in failing human myocardium is accompanied by enhanced sensitivity of the failing human myocardium toward cardiac glycosides and Na+ -channel activators.	Glycosides	197-207	solute carrier family 8 member A1	Homo sapiens	64-82	
8615321-7	1996	These results suggest that human failing myocardium exerts and enhanced sensitivity to cardiac glycosides and Na+ -channel activators, possibly because of enhanced expression of the Na+/Ca2+ exchanger or because of an altered intracellular Na+ -homeostasis.	Glycosides	95-105	solute carrier family 8 member A1	Homo sapiens	182-200	
19647009-5	2009	In human colon cancer HT29 cells both glycosides increased the [Ca2+](i) and this event was dependent on the calcium influx via the Na+/Ca2+ exchanger.	Glycosides	38-48	solute carrier family 8 member A1	Homo sapiens	132-150	
19647009-8	2009	All the effects of glycosides were prevented by inhibiting the Na+/Ca2+ exchanger or the calmodulin kinase II.	Glycosides	19-29	solute carrier family 8 member A1	Homo sapiens	63-81	
10217649-1	1999	BACKGROUND: Cardiac glycosides initiate an increase in force of contraction by inhibiting the sarcolemmal sodium pump (Na+, K+-ATPase), thereby decreasing Ca2+ extrusion by the Na+-Ca2+ exchanger, which increases the cellular content of Ca2+.	Glycosides	20-30	solute carrier family 8 member A1	Homo sapiens	177-195