PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19687166-4	2010	Accordingly, we determined the contribution of NHE-1 to cardiac glycoside-induced hypertrophy.	Glycosides	64-73	solute carrier family 9 member A1	Rattus norvegicus	47-52	
19687166-8	2010	Conversely, both glycosides increased NHE-1 expression in cardiomyocytes by approximately two-fold and significantly increased intracellular sodium concentrations by more than 60% (P < 0.05).	Glycosides	17-27	solute carrier family 9 member A1	Rattus norvegicus	38-43	
19687166-13	2010	CONCLUSION: When taken together, these studies show that NHE-1 inhibition attenuates the hypertrophic effect of cardiac glycosides without affecting inotropic parameters and suggest a possible approach to limiting glycoside-induced hypertrophic responses while preserving therapeutic, i.e. inotropic, actions.	Glycosides	120-130	solute carrier family 9 member A1	Rattus norvegicus	57-62	
19687166-13	2010	CONCLUSION: When taken together, these studies show that NHE-1 inhibition attenuates the hypertrophic effect of cardiac glycosides without affecting inotropic parameters and suggest a possible approach to limiting glycoside-induced hypertrophic responses while preserving therapeutic, i.e. inotropic, actions.	Glycosides	120-129	solute carrier family 9 member A1	Rattus norvegicus	57-62