PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31781509-8	2019	Results: Our results showed that the qurictine glycosides (PJ-1 and PJ-9) selectively inhibited the mutant K-Ras/B-Raf proteins expression and interaction in both cancer cells; while SOR showed obvious depletion of total Raf-1 protein in cancer cells and normal cells as well.	Glycosides	37-57	KRAS proto-oncogene, GTPase	Homo sapiens	107-112	
32814087-3	2020	Here we report a compound library screen and identification of cardiac glycosides as agents that selectively enhance the in vitro and in vivo effects of chemotherapy on KRAS mutant lung cancer.	Glycosides	71-81	KRAS proto-oncogene, GTPase	Homo sapiens	169-173	
32814087-5	2020	Inhibition of UHRF1 by cardiac glycosides was mediated by specific suppression of the oncogenic KRAS pathway.	Glycosides	31-41	KRAS proto-oncogene, GTPase	Homo sapiens	96-100