PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33017468-8	2020	A-1331852-induced apoptosis was most efficacious when prosurvival MCL-1 was additionally targeted, either by BH3-mimetics or genetic deletion.	A-1331852	0-9	MCL1 apoptosis regulator, BCL2 family member	Homo sapiens	66-71	
32346617-11	2020	Co-inhibition of BCL-XL and MCL-1 with A1331852 and S63845 significantly inhibited cell proliferation of all four cell lines.	A-1331852	39-47	MCL1 apoptosis regulator, BCL2 family member	Homo sapiens	28-33	
26939706-9	2016	In contrast, xenografts that expressed BCL-XL, MCL-1, and BCL-2 were more sensitive to the combination of bortezomib with a BCL-XL selective inhibitor (A-1331852) but not with venetoclax cotreatment when compared with monotherapies.	A-1331852	152-161	MCL1 apoptosis regulator, BCL2 family member	Homo sapiens	47-52