PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20947823-7	2011	Circulating haptoglobin and glutathione/total glutathione were significantly higher in the sm22alpha-GLUT1 mice postinjury compared with controls (n=4, P<0.05), suggesting increased flux through the pentose phosphate pathway.	Pentosephosphates	202-219	solute carrier family 2 (facilitated glucose transporter), member 1	Mus musculus	101-106	
24726364-4	2014	Overexpression of the glucose transporter GLUT1 in myeloid cells caused increased glycolysis and flux through the pentose phosphate pathway but did not induce cytokines.	Pentosephosphates	114-131	solute carrier family 2 (facilitated glucose transporter), member 1	Mus musculus	42-47	
26573871-4	2016	The underlying mechanisms were defined that the anticancer effects of apigenin were reversed by ectopic GLUT1 overexpression and galactose supplementation, through activation of pentose phosphate pathway-mediated NADPH generation.	Pentosephosphates	178-195	solute carrier family 2 (facilitated glucose transporter), member 1	Mus musculus	104-109	
35499042-11	2022	Specifically, elevated O-GlcNAcylation increased glucose uptake via glucose transporter 1 (GLUT1) leading to glucose metabolic flow into the pentose phosphate pathways and HBP, which regulate the metabolic reprogramming of endometrial cells.	Pentosephosphates	141-158	solute carrier family 2 (facilitated glucose transporter), member 1	Mus musculus	68-89	
35499042-11	2022	Specifically, elevated O-GlcNAcylation increased glucose uptake via glucose transporter 1 (GLUT1) leading to glucose metabolic flow into the pentose phosphate pathways and HBP, which regulate the metabolic reprogramming of endometrial cells.	Pentosephosphates	141-158	solute carrier family 2 (facilitated glucose transporter), member 1	Mus musculus	91-96	
30659108-11	2019	Together, our findings suggest that although lack of GLUT1 blunted glycolysis and the pentose phosphate pathway, MPhi were metabolically flexible enough that inflammatory cytokine release was not dramatically regulated, yet phagocytic defects hindered MPhi function in chronic diseases.	Pentosephosphates	86-103	solute carrier family 2 (facilitated glucose transporter), member 1	Mus musculus	53-58