PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31695757-14	2019	The ALDH1A1 effect on erlotinib resistance was abrogated by ROS-RCS induction and mimicked by ROS-RCS scavenging.	erlotinib	22-31	aldehyde dehydrogenase 1 family member A1	Homo sapiens	4-11	
31695757-9	2019	Results: Knockdown or pharmacological inhibition of ALDH1A1 overcame erlotinib resistance in vitro and in vivo.	erlotinib	69-78	aldehyde dehydrogenase 1 family member A1	Homo sapiens	52-59	
31695757-10	2019	ALDH1A1 overexpression was sufficient to induce erlotinib resistance.	erlotinib	48-57	aldehyde dehydrogenase 1 family member A1	Homo sapiens	0-7	
31695757-13	2019	The ALDH1A1 overexpressed cells, though resisted erlotinib, were more sensitive to SOD2 or GPX4 knockdown.	erlotinib	49-58	aldehyde dehydrogenase 1 family member A1	Homo sapiens	4-11	
31695757-18	2019	Conclusions: ALDH1A1 confers erlotinib resistance by facilitating the ROS-RCS metabolic pathway.	erlotinib	29-38	aldehyde dehydrogenase 1 family member A1	Homo sapiens	13-20	
31695757-19	2019	ALDH1A1-induced upregulation of SOD2 and GPX4, as well as ALDH1A1 itself, mitigated erlotinib-induced oxidative and carbonyl stress, and imparted the TKI resistance.	erlotinib	84-93	aldehyde dehydrogenase 1 family member A1	Homo sapiens	0-7	
31695757-19	2019	ALDH1A1-induced upregulation of SOD2 and GPX4, as well as ALDH1A1 itself, mitigated erlotinib-induced oxidative and carbonyl stress, and imparted the TKI resistance.	erlotinib	84-93	aldehyde dehydrogenase 1 family member A1	Homo sapiens	58-65