PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31695757-19	2019	ALDH1A1-induced upregulation of SOD2 and GPX4, as well as ALDH1A1 itself, mitigated erlotinib-induced oxidative and carbonyl stress, and imparted the TKI resistance.	erlotinib	84-93	superoxide dismutase 2	Homo sapiens	32-36	
31695757-11	2019	Metabolomic analysis demonstrated lower ROS-RCS levels in ALDH1A1-addicted, erlotinib-resistant cells; in line with this, key enzymes for metabolizing ROS and RCS, SOD2 and GPX4, respectively, were upregulated in these cells.	erlotinib	76-85	superoxide dismutase 2	Homo sapiens	164-168	
31695757-12	2019	Knockdown of SOD2 or GPX4 re-sensitized the resistant cells to erlotinib and the effect was abrogated by ROS-RCS scavenging and mimicked by ROS-RCS induction.	erlotinib	63-72	superoxide dismutase 2	Homo sapiens	13-17