PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26659860-4	2016	In contrast, in the presence of bafilomycin, an inhibitor of late stages of autophagy and degradation in the autophagolysosome, syncytiotrophoblasts exposed to starvation, rapamycin, or punicalagin all showed increased levels of LC3-II and p62.	bafilomycin	32-43	nucleoporin 62	Homo sapiens	240-243	
31417125-6	2019	Western blotting assays revealed decreased expression of LC3 and increased levels of p62 even after treatment with the lysosomal inhibitor bafilomycin A1, indicating impairment of the autophagic flux.	bafilomycin	139-150	nucleoporin 62	Homo sapiens	85-88	
26926601-11	2016	Blocking the terminal stage of autophagy with bafilomycin increased p62 (P = 0.01) in cells from diabetics to a lesser extent than in cells from controls (P = 0.04), suggesting ongoing, but inadequate autophagic clearance.	bafilomycin	46-57	nucleoporin 62	Homo sapiens	68-71	
32237184-9	2020	Inhibition of autophagy by bafilomycin rendered consistent expression of p62 indicating a defect in the final process of autophagy.	bafilomycin	27-38	nucleoporin 62	Homo sapiens	73-76	
26120766-6	2015	Introducing bafilomycin to Earle"s balanced salt solution or to OSS condition incrementally increased both LC3-II/LC3-I ratios and p62 levels, implicating impaired autophagic flux.	bafilomycin	12-23	nucleoporin 62	Homo sapiens	131-134	
22878401-6	2012	Bafilomycin prevented the degradation of cargo and increased LC3-II and p62 in cytotrophoblasts exposed to hypoxia, revealing enhanced autophagic flux.	bafilomycin	0-11	nucleoporin 62	Homo sapiens	72-75