PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 34651051-0 2021 Protective Mechanism of Trimetazidine in Myocardial Cells in Myocardial Infarction Rats through ERK Signaling Pathway. Trimetazidine 24-37 Eph receptor B1 Rattus norvegicus 96-99 34651051-1 2021 Objective: To study the protective effect of trimetazidine on myocardial cells in rats with myocardial infarction and explore its effect on ERK signaling pathway. Trimetazidine 45-58 Eph receptor B1 Rattus norvegicus 140-143 34651051-9 2021 Conclusion: Trimetazidine can activate ERK signaling pathway in cardiomyocytes of rats with myocardial infarction, increase the expression of p-ERK, decrease the content of ROS in cardiomyocytes, decrease the expression of apoptotic proteins, reduce myocardial infarct size, improve cardiac function, and increase myocardial function. Trimetazidine 12-25 Eph receptor B1 Rattus norvegicus 39-42 34651051-9 2021 Conclusion: Trimetazidine can activate ERK signaling pathway in cardiomyocytes of rats with myocardial infarction, increase the expression of p-ERK, decrease the content of ROS in cardiomyocytes, decrease the expression of apoptotic proteins, reduce myocardial infarct size, improve cardiac function, and increase myocardial function. Trimetazidine 12-25 Eph receptor B1 Rattus norvegicus 144-147 27121077-10 2016 Furthermore, trimetazidine inhibited phosphorylation of ERK and P38 MAPK to reduce myocardial fibrosis. Trimetazidine 13-26 Eph receptor B1 Rattus norvegicus 56-59