PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26116417-4	2015	Based on our results, the major effect from plasma exposure was found to be the activation of MAPK and p53 signaling pathways, resulting in changes in gene expression of MEKK, GADD, FOS and JUN.	gadd	176-180	tumor protein p53	Homo sapiens	103-106	
9926927-7	1999	Our results suggest that the PA26 gene is a novel p53 target gene with properties common to the GADD family of growth arrest and DNA damage-inducible stress-response genes, and, thus, a potential novel regulator of cellular growth.	gadd	96-100	tumor protein p53	Homo sapiens	50-53	
22223508-5	2014	Our data reaffirms the oncogenic capability of MIC with elevated DNA damage response proteins pATM and gamma-H2AX, deregulation of DNA damage check point genes CHK1 and CHK2, altered expression of p53 and p21 proteins involved in cell cycle arrest with perturbation in GADD-45 expression in the treated cells.	gadd	269-273	tumor protein p53	Homo sapiens	197-200	
8892753-3	1996	In contrast, the gadd gene stress response to base-damaging agents, such as methylmethane sulfonate (MMS) or UV radiation, or medium depletion (starvation) occurs in all mammalian cells examined to date regardless of p53 status for both GADD45 and also GADD153, which is not IR-responsive in many lines with functional p53.	gadd	17-21	tumor protein p53	Homo sapiens	319-322	
9927805-8	1998	The p53 gene encodes a transcription factor that contributes to several different cellular activities, including apoptosis, the cellular response to radiation, and the activation of proteins such as GADD, Bcl-2 (represses to apoptosis) and Bax.	gadd	199-203	tumor protein p53	Homo sapiens	4-7