PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21779957-3	2012	Pretreatment with potassium cyanide (KCN, a cytochrome pathway inhibitor) greatly increased CN-resistant R and reduced reactive oxygen species (ROS) formation, while application of salicylhydroxamic acid (SHAM, an AOX inhibitor) blocked the AOX activity and enhanced the production of ROS in the plants.	salicylhydroxamic acid	181-203	acyl-CoA oxidase 1	Homo sapiens	241-244	
22429403-4	2012	The inhibition of the AOX pathway by salicylhydroxamic acid (SHAM) caused the over-reduction of the photosystem I (PSI) acceptor side, as indicated by the increases in the extent of reduction of P700+.	salicylhydroxamic acid	37-59	acyl-CoA oxidase 1	Homo sapiens	22-25	
20059739-6	2010	Upon restriction of AOX pathway using salicylhydroxamic acid (SHAM), the observed decrease in NaHCO(3)-dependent O(2) evolution or p-benzoquinone (BQ)-dependent O(2) evolution [indicator of photosystem II (PSII) activity] and the increase in total cellular levels of pyruvate and malate were further aggravated/promoted under HL.	salicylhydroxamic acid	38-60	acyl-CoA oxidase 1	Homo sapiens	20-23	
9724695-0	1998	Cyanide restores N gene-mediated resistance to tobacco mosaic virus in transgenic tobacco expressing salicylic acid hydroxylase Salicylhydroxamic acid (SHAM), an inhibitor of alternative oxidase (AOX), blocks salicylic acid-induced resistance to tobacco mosaic virus (TMV) but does not inhibit pathogenesis-related PR-1 protein synthesis or resistance to fungal and bacterial pathogens.	salicylhydroxamic acid	129-151	acyl-CoA oxidase 1	Homo sapiens	197-200	
19958137-5	2010	Salicylhydroxamic acid (SHAM, an AOX inhibitor) pretreatment reduced the DEA/NO-induced cyanide-resistant respiration and partially compromised induced resistance to TMV.	salicylhydroxamic acid	0-22	acyl-CoA oxidase 1	Homo sapiens	33-36	
11351100-10	2001	Inhibition of the AOX (with salicylhydroxamic acid) decreases respiration rates less than the activity present before inhibition (i.e. measured with the 18O-fractionation technique).	salicylhydroxamic acid	28-50	acyl-CoA oxidase 1	Homo sapiens	18-21	
31795459-6	2019	However, this mitigating effect was aggravated by salicylhydroxamic acid (SHAM, an AOX inhibitor), suggesting that AP contributes to NO-enhanced Cd stress tolerance.	salicylhydroxamic acid	50-72	acyl-CoA oxidase 1	Homo sapiens	83-86	
31795459-6	2019	However, this mitigating effect was aggravated by salicylhydroxamic acid (SHAM, an AOX inhibitor), suggesting that AP contributes to NO-enhanced Cd stress tolerance.	salicylhydroxamic acid	74-78	acyl-CoA oxidase 1	Homo sapiens	83-86