PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10052739-0	1999	Effectiveness of 1,25-dihydroxyvitamin D supplementation on blood pressure reduction in a pseudohypoparathyroidism patient with high renin activity.	1,25-dihydroxyvitamin D	17-40	renin	Homo sapiens	133-138	
20065152-1	2010	Vitamin D receptors have been detected in vascular smooth muscle cells, and 1,25-dihydroxyvitamin D inhibits the renin mRNA expression.	1,25-dihydroxyvitamin D	76-99	renin	Homo sapiens	113-118	
19034301-6	2008	Our study shows that suppression of renin expression by 1,25-dihydroxyvitamin D in vivo is independent of parathyroid hormone and calcium.	1,25-dihydroxyvitamin D	56-79	renin	Homo sapiens	36-41	
20515678-0	2010	Independent association between 1,25-dihydroxyvitamin D, 25-hydroxyvitamin D and the renin-angiotensin system: The Ludwigshafen Risk and Cardiovascular Health (LURIC) study.	1,25-dihydroxyvitamin D	32-55	renin	Homo sapiens	85-90	
20515678-1	2010	BACKGROUND: Antihypertensive and tissue-protective properties of vitamin D metabolites are increasingly attributed to the inhibition of renin synthesis by 1,25-dihydroxyvitamin D [1,25(OH)2D] in the kidney.	1,25-dihydroxyvitamin D	155-178	renin	Homo sapiens	136-141	
17372031-2	2007	Laboratory studies indicate that 1,25-dihydroxyvitamin D suppresses renin expression and vascular smooth muscle cell proliferation; clinical studies demonstrate an inverse association between ultraviolet radiation, a surrogate marker for vitamin D synthesis, and blood pressure.	1,25-dihydroxyvitamin D	33-56	renin	Homo sapiens	68-73	
30057603-1	2018	Objective: 1,25-Dihydroxyvitamin D (1,25([OH]2D) is considered to be a negative endogenous regulator of the renin-angiotensin-aldosterone system (RAAS), but the effect of vitamin D supplementation on the RAAS is inconclusive.	1,25-dihydroxyvitamin D	11-34	renin	Homo sapiens	108-113	
2081010-6	1990	It is postulated that the increase in dietary sodium led to an increase in serum 1,25-dihydroxyvitamin D concentration, which may have contributed to an increase in intracellular calcium concentration, a decrease in renal secretion of renin, and a fall in plasma renin activity.	1,25-dihydroxyvitamin D	81-104	renin	Homo sapiens	235-240	
2081010-6	1990	It is postulated that the increase in dietary sodium led to an increase in serum 1,25-dihydroxyvitamin D concentration, which may have contributed to an increase in intracellular calcium concentration, a decrease in renal secretion of renin, and a fall in plasma renin activity.	1,25-dihydroxyvitamin D	81-104	renin	Homo sapiens	263-268	
2081010-8	1990	Therefore, 1,25-dihydroxyvitamin D may be a mediator in the response of high renin hypertension to increased sodium intake.	1,25-dihydroxyvitamin D	11-34	renin	Homo sapiens	77-82	
2856782-4	1985	We observed renin-linked, heterogeneous deviations in circulating levels of the divalent cations, magnesium and ionized calcium, in addition to deviations in the calcium-regulating hormones, parathyroid hormone (PTH), calcitonin (CT), and 1,25 dihydroxyvitamin D (1,25 D).	1,25-dihydroxyvitamin D	239-262	renin	Homo sapiens	12-17	
33230698-4	2021	FGF23 inhibits the activation of 1,25-dihydroxyvitamin D that is a potent suppressor of renin biosynthesis.	1,25-dihydroxyvitamin D	33-56	renin	Homo sapiens	88-93	
2081010-0	1990	Interaction of 1,25-dihydroxyvitamin D and plasma renin activity in high renin essential hypertension.	1,25-dihydroxyvitamin D	15-38	renin	Homo sapiens	50-55	
2081010-0	1990	Interaction of 1,25-dihydroxyvitamin D and plasma renin activity in high renin essential hypertension.	1,25-dihydroxyvitamin D	15-38	renin	Homo sapiens	73-78