PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27127181-2	2016	Here we observed that Dexamethasone (Dex) induces a robust cytoskeleton rearrangement and decreases Cx43 protein expression in osteocyte-like MLO-Y4 cells.	Dexamethasone	22-35	gap junction protein, alpha 3	Mus musculus	100-104	
27127181-2	2016	Here we observed that Dexamethasone (Dex) induces a robust cytoskeleton rearrangement and decreases Cx43 protein expression in osteocyte-like MLO-Y4 cells.	Dexamethasone	22-25	gap junction protein, alpha 3	Mus musculus	100-104	
27127181-4	2016	Loss of dendritic processes is a consequence of reduced Cx43 connectivity upon Dex induced autophagy in both RFP-GFP-LC3B transfected MLO-Y4 cells and primary calvarial osteocytes from LC3GFP transgenic mice.	Dexamethasone	79-82	gap junction protein, alpha 3	Mus musculus	56-60	
27127181-5	2016	Upon the induction of autophagy by Dex, Cx43 was internalized into autophagosome/autolysosomes and degraded by autophagy.	Dexamethasone	35-38	gap junction protein, alpha 3	Mus musculus	40-44	
27127181-7	2016	Inhibition Akt-mTORC1 signaling by Dex induces autophagy subsequently resulting in Cx43 degradation.Activation of Akt phosphorylation by IGF-1 attenuated Dex induced autophagy and degradation of Cx43.	Dexamethasone	35-38	gap junction protein, alpha 3	Mus musculus	83-87	
32521774-3	2020	Recently, de novo expression of connexin43 and connexin45 hemichannels (Cx43 HCs and Cx45 HCs, respectively) has been proposed to play a critical role in the mechanism underlying myofiber atrophy induced by dexamethasone (Dex: a synthetic glucocorticoid), but their involvement in specific muscle changes promoted by Dex remains poorly understood.	Dexamethasone	207-220	gap junction protein, alpha 3	Mus musculus	72-76