PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19397752-7	2009	At baseline interleukin (IL)-13 and IL-15 concentrations were significantly higher in the dexamethasone treatment group.	Dexamethasone	90-103	interleukin 15	Homo sapiens	36-41	
21706285-2	2011	In the present study, we extend our findings to methylprednisolone (MeP) and dexamethasone (Dex), thus ascribing to glucocorticoids (GCs) a general feature as positive regulators of IL-15-mediated effects on NK cells.	Dexamethasone	77-90	interleukin 15	Homo sapiens	182-187	
21706285-2	2011	In the present study, we extend our findings to methylprednisolone (MeP) and dexamethasone (Dex), thus ascribing to glucocorticoids (GCs) a general feature as positive regulators of IL-15-mediated effects on NK cells.	Dexamethasone	92-95	interleukin 15	Homo sapiens	182-187	
8816384-6	1996	Dexamethasone-pretreated cells respond to IL-15 poorly, while the response to IL-2 is not affected.	Dexamethasone	0-13	interleukin 15	Homo sapiens	42-47	
15153534-4	2004	Surprisingly, we found that incubation of unfractionated PBMC with IL-15 for 48 h resulted in the inhibition of glucocorticoid receptor (GCR) nuclear translocation in response to dexamethasone (DEX) treatment in CD19-positive B cells significantly greater than CD19-negative non-B cells (p < 0.01).	Dexamethasone	194-197	interleukin 15	Homo sapiens	67-72	
15153534-9	2004	In the presence of IL-4-neutralizing Ab, when B cells were cocultured with CD56(+) cells and IL-15, the B cells were found to be steroid sensitive, i.e., DEX induced GCR nuclear translocation.	Dexamethasone	154-157	interleukin 15	Homo sapiens	93-98	
9639524-6	1998	IL-7 and IL-15 also supported the growth of SeAx cells in the presence of the apoptosis inducing agents dexamethasone and retinoic acid.	Dexamethasone	104-117	interleukin 15	Homo sapiens	9-14	
15153534-4	2004	Surprisingly, we found that incubation of unfractionated PBMC with IL-15 for 48 h resulted in the inhibition of glucocorticoid receptor (GCR) nuclear translocation in response to dexamethasone (DEX) treatment in CD19-positive B cells significantly greater than CD19-negative non-B cells (p < 0.01).	Dexamethasone	179-192	interleukin 15	Homo sapiens	67-72	
15124245-8	2004	Dexamethasone and pyrolidine dithiocarbamate, but not curcumin, completely blocked the IL-15-induced upregulation of COX-2 mRNA.	Dexamethasone	0-13	interleukin 15	Homo sapiens	87-92	
11571455-14	2001	However, IL-15 activity is effectively inhibited by Rapa and Dex but not by CsA.	Dexamethasone	61-64	interleukin 15	Homo sapiens	9-14	
10086856-5	1999	Dexamethasone (10(-5)-10(-6) M) markedly inhibited IL-15 mRNA expression by normal and transformed keratinocytes in a range of pharmacological concentrations.	Dexamethasone	0-13	interleukin 15	Homo sapiens	51-56	
9334724-5	1997	Studying whether and how IL-15 modulates distinct apoptosis pathways, we show here that apoptosis induced by anti-Fas, anti-CD3, dexamethasone, and/or anti-IgM in activated human T and B cells in vitro is inhibited by IL-15 in a manner dependent on RNA synthesis.	Dexamethasone	129-142	interleukin 15	Homo sapiens	25-30	
29609271-7	2018	IL-15 significantly increased the proliferation and survival of Robo1-CAR-NK92 cells, but IL-21 played the opposite role.Remarkably, IL-21 and IL-15+ IL-21 enhanced the cytotoxicity of NK92 and Robo1-CAR-NK92 cells.The combination of dexamethasone and interleukins dramatically promoted the proliferation and survival but obviously impaired the cytotoxicity of NK92 and Robo1-CAR-NK92 cells (except that IL+ 15 and dexamethasone have no effect on the cytotoxicity of Robo1-CAR-NK92 cells).	Dexamethasone	234-247	interleukin 15	Homo sapiens	0-5	
29609271-7	2018	IL-15 significantly increased the proliferation and survival of Robo1-CAR-NK92 cells, but IL-21 played the opposite role.Remarkably, IL-21 and IL-15+ IL-21 enhanced the cytotoxicity of NK92 and Robo1-CAR-NK92 cells.The combination of dexamethasone and interleukins dramatically promoted the proliferation and survival but obviously impaired the cytotoxicity of NK92 and Robo1-CAR-NK92 cells (except that IL+ 15 and dexamethasone have no effect on the cytotoxicity of Robo1-CAR-NK92 cells).	Dexamethasone	234-247	interleukin 15	Homo sapiens	143-148	
29609271-7	2018	IL-15 significantly increased the proliferation and survival of Robo1-CAR-NK92 cells, but IL-21 played the opposite role.Remarkably, IL-21 and IL-15+ IL-21 enhanced the cytotoxicity of NK92 and Robo1-CAR-NK92 cells.The combination of dexamethasone and interleukins dramatically promoted the proliferation and survival but obviously impaired the cytotoxicity of NK92 and Robo1-CAR-NK92 cells (except that IL+ 15 and dexamethasone have no effect on the cytotoxicity of Robo1-CAR-NK92 cells).	Dexamethasone	415-428	interleukin 15	Homo sapiens	0-5	
29609271-7	2018	IL-15 significantly increased the proliferation and survival of Robo1-CAR-NK92 cells, but IL-21 played the opposite role.Remarkably, IL-21 and IL-15+ IL-21 enhanced the cytotoxicity of NK92 and Robo1-CAR-NK92 cells.The combination of dexamethasone and interleukins dramatically promoted the proliferation and survival but obviously impaired the cytotoxicity of NK92 and Robo1-CAR-NK92 cells (except that IL+ 15 and dexamethasone have no effect on the cytotoxicity of Robo1-CAR-NK92 cells).	Dexamethasone	415-428	interleukin 15	Homo sapiens	143-148