PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12697421-8	2003	In addition, anti-inflammatory drugs dexamethasone (DEX) and cyclosporin A (CSA) both blocked activation of JNKS/SAPK and the cell-cell contact induced production of hRPE IL-8 and MCP-1, while activation of p38 and ERK was only inhibited by DEX, but not by CSA.	Dexamethasone	37-50	mitogen-activated protein kinase 9	Homo sapiens	113-117	
12697421-8	2003	In addition, anti-inflammatory drugs dexamethasone (DEX) and cyclosporin A (CSA) both blocked activation of JNKS/SAPK and the cell-cell contact induced production of hRPE IL-8 and MCP-1, while activation of p38 and ERK was only inhibited by DEX, but not by CSA.	Dexamethasone	52-55	mitogen-activated protein kinase 9	Homo sapiens	113-117	
9343388-7	1997	Furthermore, we demonstrate that dexamethasone, which inhibits the production of cytokines, including TNF-alpha, significantly inhibits LPS induction of JNK/SAPK activity but not that of p38, ERK1 and ERK2, or MEK3, MEK4, or MEK6.	Dexamethasone	33-46	mitogen-activated protein kinase 9	Homo sapiens	157-161	
9639523-3	1998	However, IL-6-dependent protection of viability correlated with an inhibition of dexamethasone- and anti-fas-induced activation of jun kinase (JNK) and AP-1 transactivation.	Dexamethasone	81-94	mitogen-activated protein kinase 9	Homo sapiens	131-141	
9639523-3	1998	However, IL-6-dependent protection of viability correlated with an inhibition of dexamethasone- and anti-fas-induced activation of jun kinase (JNK) and AP-1 transactivation.	Dexamethasone	81-94	mitogen-activated protein kinase 9	Homo sapiens	143-146