PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
6986577-3	1980	5-Hydroxytryptophan (5-HTP) caused a significant increase (p less than 0.05) in arterial plasma renin activity (pra) that was abolished when peripheral and central aromatic amino acid decarboxylase activities were inhibited by administration of benserazide, but not reduced when only the peripheral decarboxylase activities were inhibited by administration of carbidopa.	5-Hydroxytryptophan	0-19	renin	Canis lupus familiaris	96-101	
6986577-3	1980	5-Hydroxytryptophan (5-HTP) caused a significant increase (p less than 0.05) in arterial plasma renin activity (pra) that was abolished when peripheral and central aromatic amino acid decarboxylase activities were inhibited by administration of benserazide, but not reduced when only the peripheral decarboxylase activities were inhibited by administration of carbidopa.	5-Hydroxytryptophan	21-26	renin	Canis lupus familiaris	96-101	
6986577-4	1980	The serotonin receptor blocking drug metergoline also abolished the renin response to 5-HTP.	5-Hydroxytryptophan	86-91	renin	Canis lupus familiaris	68-73	
6986577-9	1980	These data indicate that 5-HTP and L-Tryptophan act on the central nervous system to produce an increase in renin secretion that is mediated via the renal nerves and occurs without a concomitant increase in sympathetic output to the heart or blood vessels.	5-Hydroxytryptophan	25-30	renin	Canis lupus familiaris	108-113