PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19956887-13	2010	Taken together, these results indicate that eupatolide could augment TRAIL-induced apoptosis in human breast cancer cells by down-regulating c-FLIP expression through the inhibition of AKT phosphorylation and be a valuable compound to overcome TRAIL resistance in breast cancer cells.	eupatolide	44-54	AKT serine/threonine kinase 1	Homo sapiens	185-188	
29113242-7	2017	In addition to this canonical pathway, the non-canonical protein kinase B (AKT) and extracellular signal-regulated kinase (ERK) pathways were also inhibited by eupatolide treatment.	eupatolide	160-170	AKT serine/threonine kinase 1	Homo sapiens	75-78	
29113242-8	2017	In summary, the results suggest that eupatolide suppresses the migration and invasion of breast cancer cells by blocking the canonical ALK5-SMAD3 signaling pathway and the non-canonical ERK and AKT signaling pathways.	eupatolide	37-47	AKT serine/threonine kinase 1	Homo sapiens	194-197