PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30325183-11	2018	Among the multiple genes, pathways and networks that are dependent on SRC-2 during hESC decidualization, first-line analysis supports a critical role for this coregulator in maintaining retinoid signaling during progesterone-driven decidualization.	Progesterone	212-224	nuclear receptor coactivator 2	Homo sapiens	70-75	
24571987-3	2014	Although data mining revealed that only a small subset of the total progesterone-dependent transcriptional changes is dependent on SRC-2 (~13%), key genes previously reported to mediate progesterone-driven endometrial stromal cell decidualization are present within this subset.	Progesterone	68-80	nuclear receptor coactivator 2	Homo sapiens	131-136	
24571987-4	2014	Along with providing a more detailed molecular portrait of the decidual transcriptional program governed by SRC-2, the degree of functional diversity of these progesterone mediators underscores the pleiotropic regulatory role of SRC-2 in this tissue.	Progesterone	159-171	nuclear receptor coactivator 2	Homo sapiens	229-234	
24739643-21	2014	CONCLUSION: During the expression of SDF-1alpha regulated by steroids in ectopic endometrium cells, SRC-1 is the major coactivator of 17beta-estradiol and SRC-2 is the major coactivator of progesterone.	Progesterone	189-201	nuclear receptor coactivator 2	Homo sapiens	155-160	
10999746-6	2000	The expression of SRC-1 and TIF2 is significantly related to progesterone but not to estrogen receptor expression.	Progesterone	61-73	nuclear receptor coactivator 2	Homo sapiens	28-32