PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23313075-6	2013	PRL added to the CG compartment increased the ovarian release of P, estradiol, nitrites and PGF2alpha, and decreased the Bcl-2/Bax ratio in non-lactating rats; yet, with the exception of a reduction in the release of nitrites, such parameters were not modified in lactating animals.	Progesterone	0-1	BCL2, apoptosis regulator	Rattus norvegicus	121-126	
12520090-7	2002	RESULTS: The adenoviral gene transfer system induced Bcl-2 expression in more than 70% of beta-cells and the protein expression levels were successfully regulated in response to varying concentrations of progesterone antagonist RU 486.	Progesterone	204-216	BCL2, apoptosis regulator	Rattus norvegicus	53-58	
10859259-3	2000	Antiprogestin (RU486) was also administered during proliferation of DB to study progesterone regulation of Bax/Bcl2 balance.	Progesterone	80-92	BCL2, apoptosis regulator	Rattus norvegicus	111-115	
9115235-9	1997	As the low level of bcl-2 and bax mRNA was not influenced by progesterone treatment, the observed changes in total amount of bcl-X transcripts and spliced isoforms could represent the mechanism by which progesterone controls cell death in epithelial cells of the endometrium.	Progesterone	203-215	BCL2, apoptosis regulator	Rattus norvegicus	20-25