PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21602037-4	2011	In addition, DHEA counteracted activation of NFkappaB as well as the subsequent increase in TGFbeta-1 and CTGF induced by the Se- and VE-deficient diet.	Dehydroepiandrosterone	13-17	transforming growth factor, beta 1	Rattus norvegicus	92-101	
21349280-8	2011	Glucose uptake and oxidation were lower in the renal medulla of the diabetic rats treated with DHEA, and urinary TGF-beta(1), as well as total gluthatione levels, were higher in the diabetic rats treated with DHEA.	Dehydroepiandrosterone	209-213	transforming growth factor, beta 1	Rattus norvegicus	113-124	
21349280-9	2011	DHEA treatment was not beneficial to renal tissue, since it reduced the glomerular filtration rate and renal medulla metabolism, while increasing the urinary excretion of TGF-beta(1) and the compensatory response by the glutathione system, probably due to a mechanism involving a pro-oxidant action or a pro-fibrotic effect of this androgen or its derivatives.	Dehydroepiandrosterone	0-4	transforming growth factor, beta 1	Rattus norvegicus	171-182	
33811895-11	2021	The TGF-beta1 and TGF-beta2 genes associated with the TGF-beta pathway were seen to be increased in DHEA-induced PCOS rats which showed a possible relation between the two conditions.	Dehydroepiandrosterone	100-104	transforming growth factor, beta 1	Rattus norvegicus	4-13