PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30690843-6	2019	In terms of mechanism, we pretreated fibroblasts with the endoplasmic reticulum stress (ER) inhibitor salubrinal and RNA-dependent protein kinase-like ER kinase (PERK) short hairpin RNA, these treatments abolished the inhibitory effects of HCPT on fibrosis, thereby suggesting that HCPT"s inhibition of TGF-beta1-induced tendon fibrosis might be mediated by the PERK signaling pathway in vitro.	hydroxycamptothecinum	240-244	transforming growth factor beta 1	Homo sapiens	303-312