PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24103367-7	2014	Biochemical analysis in vivo revealed that intrathecal clonidine administration specifically decreased the content of GluN2B subunit-containing NMDAR at synaptosomal membrane fraction, a result associated closely with the alleviation of inflammatory pain.	Clonidine	55-64	glutamate receptor, ionotropic, NMDA2B (epsilon 2)	Mus musculus	118-124	
24374198-10	2014	Through PKA/protein phosphatase/CaMKII pathway, clonidine noticeably decreased CFA-evoked phosphorylation of N-methyl-d-aspartate subtype glutamate receptor GluN1 and GluN2B subunit as well as alpha-amino-3-hydroxy-5-methylisoxazole-4-propionic Acid subtype glutamate receptor GluA1 subunit.	Clonidine	48-57	glutamate receptor, ionotropic, NMDA2B (epsilon 2)	Mus musculus	167-173	
24103367-8	2014	Electrophysiological recordings in vitro further demonstrated that GluN2B receptor-selective inhibitor ifenprodil dramatically reduced NMDAR synaptic responses in inflamed mice and more importantly, occluded the synaptic inhibition produced by clonidine.	Clonidine	244-253	glutamate receptor, ionotropic, NMDA2B (epsilon 2)	Mus musculus	67-73