PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
35361954-4	2022	The loss of MCEE results in reduced propionate-driven anaplerotic flux and intracellular and intratumoral accumulation of methylmalonic acid, a by-product of propionate metabolism that promotes cancer cell invasiveness.	Methylmalonic Acid	122-140	methylmalonyl-CoA epimerase	Homo sapiens	12-16	
17823972-2	2007	Two patients who excrete methylmalonic acid have recently been shown to have a homozygous nonsense mutation in the gene coding for methylmalonyl CoA epimerase (MCEE).	Methylmalonic Acid	25-43	methylmalonyl-CoA epimerase	Homo sapiens	131-158	
17823972-2	2007	Two patients who excrete methylmalonic acid have recently been shown to have a homozygous nonsense mutation in the gene coding for methylmalonyl CoA epimerase (MCEE).	Methylmalonic Acid	25-43	methylmalonyl-CoA epimerase	Homo sapiens	160-164	
17823972-3	2007	To further understand the cause of methylmalonic acid excretion, the MCEE gene was sequenced in 229 patients with elevations of methylmalonic acid excretion for which no cause was known.	Methylmalonic Acid	35-53	methylmalonyl-CoA epimerase	Homo sapiens	69-73	
17823972-3	2007	To further understand the cause of methylmalonic acid excretion, the MCEE gene was sequenced in 229 patients with elevations of methylmalonic acid excretion for which no cause was known.	Methylmalonic Acid	128-146	methylmalonyl-CoA epimerase	Homo sapiens	69-73	
31146325-0	2019	MCEE Mutations in an Adult Patient with Parkinson"s Disease, Dementia, Stroke and Elevated Levels of Methylmalonic Acid.	Methylmalonic Acid	101-119	methylmalonyl-CoA epimerase	Homo sapiens	0-4