PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 17494634-5 2007 In addition, the mRNA of nuclear protein c-myc was also increased in 5-FMO- and ornithine-treated hTERT-RPE cells, and gene silencing of c-myc prevented the induction of CAT-1 and ODC. Ornithine 80-89 MYC proto-oncogene, bHLH transcription factor Homo sapiens 41-46 17494634-5 2007 In addition, the mRNA of nuclear protein c-myc was also increased in 5-FMO- and ornithine-treated hTERT-RPE cells, and gene silencing of c-myc prevented the induction of CAT-1 and ODC. Ornithine 80-89 MYC proto-oncogene, bHLH transcription factor Homo sapiens 137-142 28270654-8 2017 An ornithine metabolism imbalance resulting from overexpression of Aurora Kinase A as a result of a single, recurrent heterozygous deletion on chromosome 19, common to all fibrolamellar carcinomas, can lead to a c-Myc and ornithine decarboxylase overexpression that results in ornithine transcarboxylase dysfunction with urea cycle disorder and subsequent hyperammonemia. Ornithine 3-12 MYC proto-oncogene, bHLH transcription factor Homo sapiens 212-217