PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18498889-0	2008	Effect of 2-hydroxyethyl methacrylate on human pulp cell survival pathways ERK and AKT.	hydroxyethyl methacrylate	10-37	mitogen-activated protein kinase 1	Homo sapiens	75-78	
18498889-10	2008	Our findings demonstrated that HEMA exposure modulates the ERK and AKT pathways in different manners, and that in turn, they function in parallel to mediate pro-survival signaling in pulp cells subjected to HEMA cytotoxicity.	hydroxyethyl methacrylate	31-35	mitogen-activated protein kinase 1	Homo sapiens	59-62	
18498889-10	2008	Our findings demonstrated that HEMA exposure modulates the ERK and AKT pathways in different manners, and that in turn, they function in parallel to mediate pro-survival signaling in pulp cells subjected to HEMA cytotoxicity.	hydroxyethyl methacrylate	207-211	mitogen-activated protein kinase 1	Homo sapiens	59-62	
16430953-8	2007	RESULTS: HEMA or TEGDMA exposure resulted in ROS formation and concentration-dependent apoptosis as well as phosphorylation of ERK.	hydroxyethyl methacrylate	9-13	mitogen-activated protein kinase 1	Homo sapiens	127-130	
16430953-10	2007	Selective inhibitors of ERK and JNK modified the apoptotic response after HEMA and TEGDMA exposure, whereas p38 inhibition modified the apoptotic response only after HEMA exposure.	hydroxyethyl methacrylate	74-78	mitogen-activated protein kinase 1	Homo sapiens	24-27