PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31333785-4	2019	Despite not increasing the intercellular ROS levels in gastric cancer cells, chaetocin did cause a reduction in mitochondrial membrane potential probably through its regulation on the expression of Bcl-2 and BAX.	chaetocin	77-86	BCL2 associated X, apoptosis regulator	Homo sapiens	208-211	
28419143-7	2017	Additionally, chaetocin treatment significantly up-regulated the protein levels of Bax, cleaved caspase-9/-3, simultaneously down-regulated the protein levels of Bcl-2, procaspase-9/-3, and activated caspase-9/-3 activity in the melanoma cells.	chaetocin	14-23	BCL2 associated X, apoptosis regulator	Homo sapiens	83-86