PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12372425-0	2002	Vascular endothelial growth factor is expressed in endothelial cells isolated from skeletal muscles of nitric oxide synthase knockout mice during prazosin-induced angiogenesis.	Prazosin	146-154	vascular endothelial growth factor A	Mus musculus	0-34	
12372425-3	2002	Using such cellular fractions from skeletal muscles of C57 mice in which angiogenesis was induced by administration with the alpha(1)-adrenergic antagonist prazosin, we found the concentration of vascular endothelial growth factor (VEGF) increased in correlation to the duration of the prazosin stimulus.	Prazosin	156-164	vascular endothelial growth factor A	Mus musculus	196-230	
12372425-3	2002	Using such cellular fractions from skeletal muscles of C57 mice in which angiogenesis was induced by administration with the alpha(1)-adrenergic antagonist prazosin, we found the concentration of vascular endothelial growth factor (VEGF) increased in correlation to the duration of the prazosin stimulus.	Prazosin	156-164	vascular endothelial growth factor A	Mus musculus	232-236	
12372425-3	2002	Using such cellular fractions from skeletal muscles of C57 mice in which angiogenesis was induced by administration with the alpha(1)-adrenergic antagonist prazosin, we found the concentration of vascular endothelial growth factor (VEGF) increased in correlation to the duration of the prazosin stimulus.	Prazosin	286-294	vascular endothelial growth factor A	Mus musculus	196-230	
12372425-3	2002	Using such cellular fractions from skeletal muscles of C57 mice in which angiogenesis was induced by administration with the alpha(1)-adrenergic antagonist prazosin, we found the concentration of vascular endothelial growth factor (VEGF) increased in correlation to the duration of the prazosin stimulus.	Prazosin	286-294	vascular endothelial growth factor A	Mus musculus	232-236	
12372425-5	2002	In neuronal nitric oxide synthase (nNOS) knockout mice, the VEGF concentration was also elevated after prazosin treatment but remained almost unchanged in endothelial nitric oxide synthase (eNOS) knockout mice.	Prazosin	103-111	vascular endothelial growth factor A	Mus musculus	60-64	
25659833-4	2015	METHODS: Myocyte-specific VEGF-A (mVEGF(-/-) ) deleted mice were treated for 7-21 days with the vasodilator prazosin to produce a sustained increase in skeletal muscle blood flow.	Prazosin	108-116	vascular endothelial growth factor A	Mus musculus	26-32	
25659833-4	2015	METHODS: Myocyte-specific VEGF-A (mVEGF(-/-) ) deleted mice were treated for 7-21 days with the vasodilator prazosin to produce a sustained increase in skeletal muscle blood flow.	Prazosin	108-116	vascular endothelial growth factor A	Mus musculus	34-44	
25659833-8	2015	In response to prazosin treatment, VEGF-A mRNA was elevated within the EDL of wild-type but not mVEGF(-/-) mice.	Prazosin	15-23	vascular endothelial growth factor A	Mus musculus	35-41	
16864416-2	2006	METHODS: VEGF Trap was administered at 10 mg x kg(-1) by subcutaneous injection twice weekly to mice, which were subject to one of two established angiogenic stimuli: (1) increasing blood flow by administration of prazosin (50 mg L(-1)); (2) muscle overload by extirpation of a synergist.	Prazosin	214-222	vascular endothelial growth factor A	Mus musculus	9-13	
27261201-8	2016	Of the dominant signalling pathways, prazosin decreased muscle VEGF, but increased its cognate receptor Flk-1 (both P < 0.01); levels of eNOS varied with blood flow (P < 0.05), and Ang-1 initially increased, while its receptor Tie-2 was unchanged, with only modest changes in the antiangiogenic factor TSP-1.	Prazosin	37-45	vascular endothelial growth factor A	Mus musculus	63-67