PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23691077-6	2013	Reserpine that exhausted cardiac NE without markedly decreasing plasma NE level abrogated the inhibitory effects of YHB on cardiac TNF-alpha and iNOS expression as well as cardiac dysfunction, but not the suppressive effects of YHB on plasma TNF-alpha and NO elevation in LPS-challenged mice.	Yohimbine	116-119	tumor necrosis factor	Mus musculus	131-140	
33220278-9	2021	DEX inhibited LPS-induced TNFalpha, IL-6, and PGE2 productions and COX-2 mRNA expression, and the effects of DEX were reversed by yohimbine.	Yohimbine	130-139	tumor necrosis factor	Mus musculus	26-34	
23691077-4	2013	Here, we demonstrated that YHB decreased nitric oxide (NO) and tumor necrosis factor-alpha (TNF-alpha) levels in the myocardium and plasma, attenuated cardiac and hepatic dysfunction, but not kidney and lung injuries in endotoxemic mice.	Yohimbine	27-30	tumor necrosis factor	Mus musculus	63-90	
23691077-11	2013	YHB-elevated cardiac NE improves cardiac function via suppressing cardiac iNOS and TNF-alpha expression, activating beta1-AR and inhibiting cardiomyocyte apoptosis through alpha1- and beta2-AR in endotoxemic mice.	Yohimbine	0-3	tumor necrosis factor	Mus musculus	83-92	
23691077-4	2013	Here, we demonstrated that YHB decreased nitric oxide (NO) and tumor necrosis factor-alpha (TNF-alpha) levels in the myocardium and plasma, attenuated cardiac and hepatic dysfunction, but not kidney and lung injuries in endotoxemic mice.	Yohimbine	27-30	tumor necrosis factor	Mus musculus	92-101	
20926983-5	2011	LPS-provoked cardiac apoptosis, I-[kappa]B[alpha] phosphorylation, IL-1[beta], TNF-[alpha], and NO production were attenuated by pretreatment with Ber and/or yohimbine, whereas cardiac Toll-like receptor 4 mRNA expression, malondialdehyde content, and superoxide dismutase activity were not affected.	Yohimbine	158-167	tumor necrosis factor	Mus musculus	79-89