PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24062160-4	2014	Analysis of the infected plants revealed that the mutant p19-3M VSR is able to induce miR168 level controlling the accumulation of the antiviral AGO1, and this activity is associated with the enhanced accumulation of viral RNAs.	mir168	86-92	interleukin 23 subunit alpha	Homo sapiens	57-60	
24062160-6	2014	Our data indicate that p19 VSR possesses two independent silencing suppressor functions, viral siRNA binding and the miR168-mediated AGO1 control, both of which are required to efficiently cope with the RNA-silencing based host defence.	mir168	117-123	interleukin 23 subunit alpha	Homo sapiens	23-26	
20823831-3	2010	We also show that the induction of AGO1 mRNA level is a part of the host defence reaction, whereas the induction of miR168, which overlaps spatially with virus-occupied sectors, is mediated mainly by the Tombusvirus p19 RNA-silencing suppressor.	mir168	116-122	interleukin 23 subunit alpha	Homo sapiens	216-219	
20823831-4	2010	The absence of p19 results in the elimination of miR168 induction and accompanied with the enhanced accumulation of AGO1 protein.	mir168	49-55	interleukin 23 subunit alpha	Homo sapiens	15-18	
20823831-5	2010	In transient expression study, p19 mediates the induction of miR168 and the down-regulation of endogenous AGO1 level.	mir168	61-67	interleukin 23 subunit alpha	Homo sapiens	31-34