PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8596046-1	1996	The present study was conducted to determine whether endogenous IL-1 is involved as a potent mediator of PGE2-stimulated osteoclast formation in 1 alpha,25-dihydroxyvitamin D3 (1 alpha,25-(OH)2D3)-primed calvarial cells from mouse embryos.	Calcitriol	145-175	interleukin 1 complex	Mus musculus	64-68	
9091574-5	1997	IL-18 inhibited OCL formation in the presence of osteoclastogenic agents including 1alpha,25-dihydroxyvitamin D3, prostaglandin E2, parathyroid hormone, IL-1, and IL-11.	Calcitriol	83-112	interleukin 1 complex	Mus musculus	0-4	
8163655-6	1994	Further, an antibody neutralizing IL-11 suppressed osteoclast development induced by either 1,25-dihydroxyvitamin D3, parathyroid hormone, interleukin-1, or tumor necrosis factor; whereas inhibitors of IL-1 or TNF had no effect on IL-11-stimulated osteoclast formation.	Calcitriol	92-116	interleukin 1 complex	Mus musculus	34-38	
8473513-0	1993	IL-1-induced murine osteoblast IL-6 production is mediated by the type 1 IL-1 receptor and is increased by 1,25 dihydroxyvitamin D3.	Calcitriol	107-131	interleukin 1 complex	Mus musculus	0-4	
1838853-6	1991	The PGE2 response to IL-1 and PTH was not affected by 1,25-(OH)2D3 at 24 h, but at 48 h 1,25-(OH)2D3 (10(-8) M) increased the PGE2 response to both IL-1 and PTH.	Calcitriol	88-100	interleukin 1 complex	Mus musculus	21-25	
3879808-2	1985	Physiological concentrations of 1,25(OH)2D3 inhibited thymocyte proliferation induced by IL-1 and IL-2 in similar fashion suggesting an inhibition of the response to IL-2 by this hormone.	Calcitriol	32-43	interleukin 1 complex	Mus musculus	89-93	
3259542-3	1988	The production was not affected, but 1,25(OH)2D3 (greater than 10(-11) M) and a synthetic derivative MC 903 (greater than = 10(-10) M) inhibited the proliferation of mouse thymocytes to IL-1.	Calcitriol	37-48	interleukin 1 complex	Mus musculus	186-190