PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21292568-11	2011	The method was applied to simultaneously determine the Sph and S1P levels in mouse kidney, human plasma, and HEK 293 cells treated with tumor necrosis factor-alpha (TNF-alpha) and N,N-dimethylsphingosine (DMS).	N,N-dimethylsphingosine	180-203	sphingosine-1-phosphate receptor 1	Mus musculus	63-66	
21292568-12	2011	The S1P levels increased in cells treated with TNF-alpha whereas decreased in cells treated with DMS.	N,N-dimethylsphingosine	97-100	sphingosine-1-phosphate receptor 1	Mus musculus	4-7	
16831409-1	2006	OBJECTIVE: N, N-Dimethylsphingosine (DMS) is recognized as an inhibitor of sphingosine kinase (SphK), a key enzyme responsible for the formation of sphingosine-1-phosphate (S1P).	N,N-dimethylsphingosine	11-35	sphingosine-1-phosphate receptor 1	Mus musculus	173-176	
18359884-7	2008	DMS or SK-I inhalation resulted in a decrease in S1P amounts in BAL fluid to basal levels, accompanied by decreased eosinophil infiltration and peroxidase activity.	N,N-dimethylsphingosine	0-3	sphingosine-1-phosphate receptor 1	Mus musculus	49-52	
15659717-6	2005	Interestingly, N,N-dimethylsphingosine, an inhibitor of SPH kinase (SK), abolished the effect of supplemented SPH but not that of S1P, suggesting that SPH acts through its conversion to S1P.	N,N-dimethylsphingosine	15-38	sphingosine-1-phosphate receptor 1	Mus musculus	186-189