PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10403658-0	1999	The UDP-glucuronyltransferase inducers, phenobarbital and pregnenolone-16alpha-carbonitrile, enhance thyroid-follicular cell apoptosis: association with TGF-beta1 expression.	Pregnenolone Carbonitrile	58-91	transforming growth factor, beta 1	Rattus norvegicus	153-162	
10403658-12	1999	In PB- and PCN-treated rats, a moderate increase in apoptosis coincided with similar increases in TGF-beta1 immunoreactive thyroid-follicular cells.	Pregnenolone Carbonitrile	11-14	transforming growth factor, beta 1	Rattus norvegicus	98-107	
10403658-13	1999	In summary, PB and PCN increase apoptosis and the percentage of TGF-beta1 positive thyroid-follicular cells.	Pregnenolone Carbonitrile	19-22	transforming growth factor, beta 1	Rattus norvegicus	64-73	
9928666-7	1998	The present study was designed to examine the dose-response effect of TSH-increasing (PB and PCN) and nonincreasing (3MC and PCB) UDP-GT inducers on apoptosis and TGF-beta1.	Pregnenolone Carbonitrile	93-96	transforming growth factor, beta 1	Rattus norvegicus	163-172	
9928666-6	1998	In a previous study in our laboratory, rats were treated for various times (up to 90 days) with PB and PCN, which increased TGF-beta1 protein and apoptosis.	Pregnenolone Carbonitrile	103-106	transforming growth factor, beta 1	Rattus norvegicus	124-133	
9928666-8	1998	PB and PCN, UDP-GT inducing compounds which increase serum TSH, increased the percentage of TGF-beta1-positive follicular cells and increased apoptosis.	Pregnenolone Carbonitrile	7-10	transforming growth factor, beta 1	Rattus norvegicus	92-101