PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26544184-5	2015	The decreased flux of glucose associated to caffeic acid affected HG induced apoptosis by down-regulating the expression of initiator (caspase 8 and 9) and effector caspases (caspase 7 and 3) and by increasing the levels of phosphorylated Bcl-2.	caffeic acid	44-56	BCL2 apoptosis regulator	Homo sapiens	239-244	
21199742-7	2010	Caffeic acid decreased levels of uncleaved caspase-3 and Bcl-2, and induced cleaved caspase-3 and p53.	caffeic acid	0-12	BCL2 apoptosis regulator	Homo sapiens	57-62	
21199742-8	2010	CONCLUSION: Caffeic acid induces apoptosis by inhibiting Bcl-2 activity, leading to release of cytochrome c and subsequent activation of caspase-3, indicating that caffeic acid induces apoptosis via the mitochondrial apoptotic pathway.	caffeic acid	12-24	BCL2 apoptosis regulator	Homo sapiens	57-62	
21199742-8	2010	CONCLUSION: Caffeic acid induces apoptosis by inhibiting Bcl-2 activity, leading to release of cytochrome c and subsequent activation of caspase-3, indicating that caffeic acid induces apoptosis via the mitochondrial apoptotic pathway.	caffeic acid	164-176	BCL2 apoptosis regulator	Homo sapiens	57-62	
16459021-0	2006	Anti-apoptotic activity of caffeic acid, ellagic acid and ferulic acid in normal human peripheral blood mononuclear cells: a Bcl-2 independent mechanism.	caffeic acid	27-39	BCL2 apoptosis regulator	Homo sapiens	125-130