PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31502574-7	2019	Additionally, the expression levels of c-Met, MMP9 and p-AKT were inhibited by calycosin in c-Met overexpressed cells.	7,3'-dihydroxy-4'-methoxyisoflavone	79-88	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	39-44	
31502574-0	2019	Calycosin down-regulates c-Met to suppress development of glioblastomas.	7,3'-dihydroxy-4'-methoxyisoflavone	0-9	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	25-30	
31502574-2	2019	In this study we focused on revealing c-Met as a potential target of calycosin suppressing glioblastomas.	7,3'-dihydroxy-4'-methoxyisoflavone	69-78	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	38-43	
31502574-4	2019	Under treatment of calycosin, the mRNA expression levels of Dtk, c-Met, Lyn and PYK2 were observed in U87 cells.	7,3'-dihydroxy-4'-methoxyisoflavone	19-28	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	65-70	
31502574-5	2019	Meanwhile a western blot assay showed that c-Met together with matrix metalloproteinases-9 (MMP9) and phosphorylation of the serine/threonine kinase AKT (p-AKT) was significantly down-regulated by calycosin.	7,3'-dihydroxy-4'-methoxyisoflavone	197-206	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	43-48	
31502574-7	2019	Additionally, the expression levels of c-Met, MMP9 and p-AKT were inhibited by calycosin in c-Met overexpressed cells.	7,3'-dihydroxy-4'-methoxyisoflavone	79-88	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	92-97	
31502574-9	2019	These data collectively indicated that calycosin possibility targeting on c-Met and exert an anti-tumor role via MMP9 and AKT.	7,3'-dihydroxy-4'-methoxyisoflavone	39-48	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	74-79