PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20540939-10	2010	Results obtained with L-NNA, 1400W, 7-NI, OxyHb, ODQ or Tiron showed that this response was mediated by products from endothelial NOS (eNOS) different from NO and without soluble guanylate cyclase activation, but it involved superoxide anions.	N-((3-(aminomethyl)phenyl)methyl)ethanimidamide	29-34	nitric oxide synthase 3	Homo sapiens	118-133	
11040050-9	2000	High iNOS versus eNOS selectivity was found for 1400W, whereas several isothiourea derivatives and 1400W displayed moderate n- versus eNOS selectivity.	N-((3-(aminomethyl)phenyl)methyl)ethanimidamide	99-104	nitric oxide synthase 3	Homo sapiens	134-138	
9030556-7	1997	Thus, 1400W was at least 5000-fold selective for iNOS versus eNOS.	N-((3-(aminomethyl)phenyl)methyl)ethanimidamide	6-11	nitric oxide synthase 3	Homo sapiens	61-65	
29471036-8	2019	The specific iNOS inhibitor 1400 W decreases eNOS S-nitrosylation and the association of eNOS and beta-catenin, thereby blocking the beta-catenin signal pathway to alleviate OxLDL-induced endothelial dysfunction.	N-((3-(aminomethyl)phenyl)methyl)ethanimidamide	28-34	nitric oxide synthase 3	Homo sapiens	45-49	
29471036-8	2019	The specific iNOS inhibitor 1400 W decreases eNOS S-nitrosylation and the association of eNOS and beta-catenin, thereby blocking the beta-catenin signal pathway to alleviate OxLDL-induced endothelial dysfunction.	N-((3-(aminomethyl)phenyl)methyl)ethanimidamide	28-34	nitric oxide synthase 3	Homo sapiens	89-93