PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24478451-3	2014	Phosphatidylinositol 3,5-bisphosphate (PI(3,5)P(2)), an endosomal signaling lipid, is implicated in insulin-dependent stimulation of TORC1 activity in adipocytes.	phosphatidylinositol 3,5-diphosphate	0-37	CREB regulated transcription coactivator 1	Homo sapiens	133-138	
24478451-8	2014	Of importance, we find that PI(3,5)P(2) is required for multiple downstream pathways via TORC1-dependent phosphorylation of additional targets, including Atg13, the modification of which inhibits autophagy, and phosphorylation of Npr1, which releases its inhibitory function and allows nutrient-dependent endocytosis.	phosphatidylinositol 3,5-diphosphate	28-39	CREB regulated transcription coactivator 1	Homo sapiens	89-94	
33157024-5	2021	Accordingly, TORC1 can phosphorylate Fab1 proximal to its PI3P-interacting FYVE domain, which causes Fab1 to shift to signaling endosomes, where it generates PI(3,5)P2.	phosphatidylinositol 3,5-diphosphate	158-167	CREB regulated transcription coactivator 1	Homo sapiens	13-18