PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33316781-7	2020	We conclude that circ_0005198-miR-198-TRIM14 regulatory pathway is critical to TMZ resistance of glioma.	Temozolomide	79-82	microRNA 198	Homo sapiens	30-37	
32813021-1	2021	BACKGROUND: Our previous studies have indicated that miR-198 reduces cellular methylguanine DNA methyltransferase (MGMT) levels to enhance temozolomide sensitivity.	Temozolomide	139-151	microRNA 198	Homo sapiens	53-60	
33316781-0	2020	Circ_0005198 enhances temozolomide resistance of glioma cells through miR-198/TRIM14 axis.	Temozolomide	22-34	microRNA 198	Homo sapiens	70-77	
33316781-6	2020	Moreover, TRIM14 was a target of miR-198 and silencing of TRIM14 hindered TMZ resistance and suppressed the progression of TMZ-resistant glioma cells, while TRIM14 over-expression rescued the inhibiting effect of miR-198 over-expression.	Temozolomide	74-77	microRNA 198	Homo sapiens	33-40	
33316781-6	2020	Moreover, TRIM14 was a target of miR-198 and silencing of TRIM14 hindered TMZ resistance and suppressed the progression of TMZ-resistant glioma cells, while TRIM14 over-expression rescued the inhibiting effect of miR-198 over-expression.	Temozolomide	123-126	microRNA 198	Homo sapiens	33-40	
28425046-8	2017	Furthermore, overexpression of MGMT restored miR-198-induced chemosensitivity to temozolomide.	Temozolomide	81-93	microRNA 198	Homo sapiens	45-52	
28425046-0	2017	MiR-198 enhances temozolomide sensitivity in glioblastoma by targeting MGMT.	Temozolomide	17-29	microRNA 198	Homo sapiens	0-7	
28425046-6	2017	And overexpression of miR-198 increased chemosensitivity to temozolomide in vitro and in vivo.	Temozolomide	60-72	microRNA 198	Homo sapiens	22-29