PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 24594322-4 2014 Here, we studied the peritoneal macrophage pattern induced by liposomes comprised of dipalmitoylphosphatidylcholine (DPPC) and cholesterol (Chol) carrying ovalbumin (OVA) (Lp DPPC/OVA), and the involvement of B-1 cells in macrophage polarization. Cholesterol 127-138 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 155-164 28726304-5 2017 Cholesterol-modified DNA associated with OVA and denaturation of OVA using urea increases the interaction. Cholesterol 0-11 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 41-44 28726304-5 2017 Cholesterol-modified DNA associated with OVA and denaturation of OVA using urea increases the interaction. Cholesterol 0-11 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 65-68 28726304-3 2017 In this study, a hydrophobic interaction-based sustained release system of ovalbumin (OVA), a model antigen, from immunostimulatory CpG DNA hydrogel is developed by the use of cholesterol-modified DNA and urea-denatured OVA (udOVA). Cholesterol 176-187 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 75-84 28726304-3 2017 In this study, a hydrophobic interaction-based sustained release system of ovalbumin (OVA), a model antigen, from immunostimulatory CpG DNA hydrogel is developed by the use of cholesterol-modified DNA and urea-denatured OVA (udOVA). Cholesterol 176-187 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 86-89 28726304-3 2017 In this study, a hydrophobic interaction-based sustained release system of ovalbumin (OVA), a model antigen, from immunostimulatory CpG DNA hydrogel is developed by the use of cholesterol-modified DNA and urea-denatured OVA (udOVA). Cholesterol 176-187 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 220-223 17234372-6 2007 Moreover, the treatment with high-density OVA-liposome (10 microg OVA in 80 nmol DSPC and 40 nmol cholesterol-liposome/20 g mouse) not only strongly suppressed IgE levels but also reduced IgG production after the boost of OVA-sensitized mice suggesting the importance of liposomal characteristic in desensitization immunotherapy. Cholesterol 98-109 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 42-45 17234372-6 2007 Moreover, the treatment with high-density OVA-liposome (10 microg OVA in 80 nmol DSPC and 40 nmol cholesterol-liposome/20 g mouse) not only strongly suppressed IgE levels but also reduced IgG production after the boost of OVA-sensitized mice suggesting the importance of liposomal characteristic in desensitization immunotherapy. Cholesterol 98-109 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 66-69 17234372-6 2007 Moreover, the treatment with high-density OVA-liposome (10 microg OVA in 80 nmol DSPC and 40 nmol cholesterol-liposome/20 g mouse) not only strongly suppressed IgE levels but also reduced IgG production after the boost of OVA-sensitized mice suggesting the importance of liposomal characteristic in desensitization immunotherapy. Cholesterol 98-109 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 66-69 15316134-3 2004 Following sensitization and inhalation exposure to ovalbumin, the bronchoalveolar lavage fluid of mice in the cholesterol group contained higher numbers of eosinophils and elevated levels of IL-5, PGE(2), and MCP-1. Cholesterol 110-121 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 51-60 15316134-7 2004 Although dietary cholesterol did not alter baseline IL-12 in the lungs, in mice challenged with ovalbumin the IL-12 levels were reduced in the cholesterol group and elevated significantly in the pravastatin group. Cholesterol 17-28 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 96-105 10486434-2 1999 Ovalbumin was encapsulated in various lyophilized niosome preparations consisting of sucrose esters, cholesterol and dicetyl phosphate. Cholesterol 101-112 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 0-9 15316134-7 2004 Although dietary cholesterol did not alter baseline IL-12 in the lungs, in mice challenged with ovalbumin the IL-12 levels were reduced in the cholesterol group and elevated significantly in the pravastatin group. Cholesterol 143-154 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 96-105 12121129-6 2002 In addition, when the liposomes with four different lipid compositions were used, OVA-liposome conjugates made using liposomes that did not contain cholesterol induced significantly higher levels of anti-OVA IgG antibody production than did those made using liposomes that contained cholesterol and, further, induced significant production of anti-OVA IgE. Cholesterol 283-294 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 82-85 11574755-6 2001 OVA aerosol-induced inflammation was significantly enhanced by dietary supplementation of 1% or 2% cholesterol. Cholesterol 99-110 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 0-3 11574755-7 2001 RESULTS: Among OVA-challenged mice, leukocyte numbers, particularly those of eosinophils, in the bronchoalveolar space increased by 3- to 5-fold with the cholesterol supplement. Cholesterol 154-165 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 15-18 11574755-8 2001 Among OVA aerosol-challenged mice, the levels of interleukin-5 and cysteinyl leukotrienes in the bronchoalveolar lavage fluid were significantly higher in those fed the 2% cholesterol diet compared with mice on the control diet. Cholesterol 172-183 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 6-9 9771618-4 1998 In contrast, liposomes prepared from distearoylphosphatidylcholine and cholesterol (6:3.5 molar ratio) were more stable: about 50% of the lipid remained as liposomes after a 4-h incubation at 37 degrees C and intact ovalbumin could be demonstrated therein by immunoblotting. Cholesterol 71-82 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 216-225 9520297-6 1997 To assess this, the highly immunogenic protein ovalbumin was conjugated onto liposomes composed of distearoylphosphatidylcholine/cholesterol (DSPC/Chol) with sufficient poly(ethylene glycol)-modified distearoyl phosphatidylethanolamine (PEG-DSPE) (2 mol%) to prevent liposome aggregation during protein coupling and to engender increased circulation lifetimes. Cholesterol 129-140 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 47-56