PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
35500641-10	2022	The proapoptotic markers (cyt c, caspase-3/-9, and Bax) were also attenuated and the antiapoptotic Bcl2 marker was increased by the preadministration of octreotide.	Octreotide	153-163	BCL2 associated X, apoptosis regulator	Rattus norvegicus	51-54	
15046398-0	2004	The effect of octreotide and bromocriptine on expression of a pro-apoptotic Bax protein in rat prolactinoma.	Octreotide	14-24	BCL2 associated X, apoptosis regulator	Rattus norvegicus	76-79	
31173973-0	2019	Inhibitory effects of octreotide on the progression of hepatic fibrosis via the regulation of Bcl-2/Bax and PI3K/AKT signaling pathways.	Octreotide	22-32	BCL2 associated X, apoptosis regulator	Rattus norvegicus	100-103	
31173973-9	2019	In summary, our study corroborated that octreotide could prevent liver fibrosis probably via modulating Bcl-2/Bax and PI3K/AKT signaling pathway.	Octreotide	40-50	BCL2 associated X, apoptosis regulator	Rattus norvegicus	110-113	
15161035-11	2004	A combination of LPS and octreotide produced a similar effect with the exception of a late increase of LICE expression, probably caused by a late increase of bax and bcl-xS.	Octreotide	25-35	BCL2 associated X, apoptosis regulator	Rattus norvegicus	158-161	
22166920-10	2012	At 12 h after operation, the expression level of Bax protein in baicalin treated group was significantly higher than those in model control group and octreotide treated group (P < 0.05 and P < 0.01, respectively).	Octreotide	150-160	BCL2 associated X, apoptosis regulator	Rattus norvegicus	49-52	
19387806-6	2009	(3) the expression of Bax protein was upregulated in pancreas and lung but downregulated in spleen and lymph nodes (P < 0.05 or P < 0.01) in Octreotide treated group; The apoptosis indexes significantly increased in lymph nodes and spleen in Octreotide treated group (P < 0.05 or P < 0.01).	Octreotide	147-157	BCL2 associated X, apoptosis regulator	Rattus norvegicus	22-25	
19387806-6	2009	(3) the expression of Bax protein was upregulated in pancreas and lung but downregulated in spleen and lymph nodes (P < 0.05 or P < 0.01) in Octreotide treated group; The apoptosis indexes significantly increased in lymph nodes and spleen in Octreotide treated group (P < 0.05 or P < 0.01).	Octreotide	248-258	BCL2 associated X, apoptosis regulator	Rattus norvegicus	22-25	
19030211-7	2008	CONCLUSION: Both baicalin and octreotide can protect rats with SAP by decreasing the contents of ALT, AST and expression levels of Bcl-2 protein, and improving the expression levels of Bax protein, caspase-3 protein, and inducing apoptosis.	Octreotide	30-40	BCL2 associated X, apoptosis regulator	Rattus norvegicus	185-188	
15046398-13	2004	It has been found that the treatment with OCT and BC, alone or in combination, causes a significant increase in Bax expression in the rat prolactinoma cells.	Octreotide	42-45	BCL2 associated X, apoptosis regulator	Rattus norvegicus	112-115	
15046398-14	2004	Our findings indicate that anti-tumoral action of bromocriptine and to some extent the action of octreotide in the experimental rat prolactinoma is connected with the induction of apoptosis and is associated with increased Bax expression.	Octreotide	97-107	BCL2 associated X, apoptosis regulator	Rattus norvegicus	223-226