PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9449644-2	1998	TNF induces receptor-mediated activation of sphingomyelinase, which converts sphingomyelin to ceramide, a mediator of TNF actions.	Ceramides	94-102	tumor necrosis factor-like	Rattus norvegicus	0-3	
22973882-0	2012	Ceramide sphingolipid signaling mediates Tumor Necrosis Factor (TNF)-dependent toxicity via caspase signaling in dopaminergic neurons.	Ceramides	0-8	tumor necrosis factor-like	Rattus norvegicus	41-62	
9449644-2	1998	TNF induces receptor-mediated activation of sphingomyelinase, which converts sphingomyelin to ceramide, a mediator of TNF actions.	Ceramides	94-102	tumor necrosis factor-like	Rattus norvegicus	118-121	
9449644-12	1998	The addition of TNF increased both the sphingomyelin and ceramide levels 3- to 5-fold in young and old cells.	Ceramides	57-65	tumor necrosis factor-like	Rattus norvegicus	16-19	
9252504-9	1997	Ceramide generated during TNF signaling leads to increased production of ROS in mitochondria.	Ceramides	0-8	tumor necrosis factor-like	Rattus norvegicus	26-29	
9111045-2	1997	Ceramide is a sphingolipid that is generated in the signaling of inflammatory cytokines such as tumor necrosis factor (TNF), which exerts many functional roles depending on the cell type where it is produced.	Ceramides	0-8	tumor necrosis factor-like	Rattus norvegicus	96-117	
9111045-2	1997	Ceramide is a sphingolipid that is generated in the signaling of inflammatory cytokines such as tumor necrosis factor (TNF), which exerts many functional roles depending on the cell type where it is produced.	Ceramides	0-8	tumor necrosis factor-like	Rattus norvegicus	119-122	
9111045-11	1997	Mitochondria isolated from TNF-treated cells showed an increase (2-3-fold) in the amount of ceramide compared with mitochondria from untreated cells.	Ceramides	92-100	tumor necrosis factor-like	Rattus norvegicus	27-30	
9111045-12	1997	These results suggest that mitochondria are a target of ceramide produced in the signaling of TNF whose effect on mitochondrial electron transport chain leads to overproduction of hydrogen peroxide and consequently this phenomena may account for the generation of reactive oxygen species during TNF cytotoxicity.	Ceramides	56-64	tumor necrosis factor-like	Rattus norvegicus	94-97	
9111045-12	1997	These results suggest that mitochondria are a target of ceramide produced in the signaling of TNF whose effect on mitochondrial electron transport chain leads to overproduction of hydrogen peroxide and consequently this phenomena may account for the generation of reactive oxygen species during TNF cytotoxicity.	Ceramides	56-64	tumor necrosis factor-like	Rattus norvegicus	295-298