PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32233204-5	2019	The TP-treated cells showed a significantly decreased p-AMPK/AMPK ratio in comparison with the control (P < 0.01), but an increased p-Akt/Akt ratio (P < 0.05) and an elevated level of p-mTOR phosphorylation (P < 0.05).	triptolide	4-6	mechanistic target of rapamycin kinase	Mus musculus	186-190	
33788266-8	2021	KEY RESULTS: Triptolide (10-100 fM) upregulated protein synthesis signals (IGF-1/p-IGF-1R/IRS-1/p-Akt/p-mTOR) and downregulated protein degradation signal atrogin-1 in C2C12 myotubes.	triptolide	13-23	mechanistic target of rapamycin kinase	Mus musculus	104-108	
32233204-6	2019	CONCLUSIONS: Triptolide can decrease the activity and induce the apoptosis of TM3 Leydig cells, and meanwhile inhibit the AMPK and activate the Akt/mTOR pathway.	triptolide	13-23	mechanistic target of rapamycin kinase	Mus musculus	148-152